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What Is the Lowest Temperature My Air Conditioning Will Read When It Is 102 Out

Increased body temperature due to an inflammatory response

Medical condition

Fever
Other names Pyrexia, febrile response, febrile[i]
Clinical thermometer 38.7.JPG
An analog medical thermometer showing a temperature of 38.7 °C or 101.seven °F
Specialty Communicable diseases, pediatrics
Symptoms Initially: shivering, feeling cold, chills[2]
Later: flushed, sweating[3]
Complications Delirious seizure[4]
Causes Virus, bacteria, increment in the trunk's temperature set point[5] [6]
Diagnostic method Temperature > between 37.ii and 38.iii °C (99.0 and 100.ix °F)[1] [seven] [8]
Differential diagnosis Hyperthermia[one]
Treatment Based on underlying cause, not required for fever itself[2] [ix]
Medication Ibuprofen, paracetamol (acetaminophen)[9] [10]
Frequency Common[two] [11]

Fever, also referred to as pyrexia, is defined as having a temperature to a higher place the normal range due to an increment in the body's temperature fix betoken.[v] [6] [vii] There is non a single agreed-upon upper limit for normal temperature with sources using values between 37.2 and 38.3 °C (99.0 and 100.9 °F) in humans.[1] [7] [8] The increase in fix point triggers increased muscle contractions and causes a feeling of cold or chills.[two] This results in greater oestrus product and efforts to conserve heat.[three] When the set point temperature returns to normal, a person feels hot, becomes flushed, and may brainstorm to sweat.[3] Rarely a fever may trigger a delirious seizure, with this being more common in young children.[iv] Fevers practice not typically go higher than 41 to 42 °C (105.8 to 107.6 °F).[6]

A fever can exist caused past many medical conditions ranging from not-serious to life-threatening.[12] This includes viral, bacterial, and parasitic infections—such as influenza, the mutual cold, meningitis, urinary tract infections, appendicitis, COVID-19, and malaria.[12] [13] Non-infectious causes include vasculitis, deep vein thrombosis, connective tissue disease, side furnishings of medication or vaccination, and cancer.[12] [xiv] Information technology differs from hyperthermia, in that hyperthermia is an increase in body temperature over the temperature set point, due to either too much rut production or non enough heat loss.[1]

Handling to reduce fever is generally not required.[two] [9] Treatment of associated pain and inflammation, nevertheless, may be useful and help a person residue.[9] Medications such as ibuprofen or paracetamol (acetaminophen) may assistance with this too every bit lower temperature.[nine] [ten] Children younger than three months require medical attention, equally might people with serious medical problems such as a compromised immune system or people with other symptoms.[xv] Hyperthermia does require treatment.[2]

Fever is i of the most common medical signs.[2] It is function of almost 30% of healthcare visits past children[2] and occurs in upward to 75% of adults who are seriously ill.[eleven] While fever evolved every bit a defense mechanism, treating fever does not appear to worsen outcomes.[16] [17] Fever is often viewed with greater concern past parents and healthcare professionals than is usually deserved, a phenomenon known every bit fever phobia.[2] [eighteen]

Associated symptoms [edit]

A fever is normally accompanied by sickness behavior, which consists of lethargy, depression, loss of appetite, sleepiness, hyperalgesia, and the inability to concentrate. Sleeping with a fever tin often cause intense or confusing nightmares, usually chosen "fever dreams".[19] Mild to severe delirium (which tin can as well cause hallucinations) may too present itself during high fevers.[xx]

Diagnosis [edit]

A range for normal temperatures has been plant.[8] Central temperatures, such as rectal temperatures, are more accurate than peripheral temperatures.[26] Fever is by and large agreed to be present if the elevated temperature is caused past a raised set indicate and:

  • Temperature in the anus (rectum/rectal) is at or over 37.5–38.3 °C (99.v–100.9 °F)[i] [8] An ear (tympanic) or forehead (temporal) temperature may also be used.[27] [28]
  • Temperature in the mouth (oral) is at or over 37.2 °C (99.0 °F) in the morning time or over 37.7 °C (99.9 °F) in the afternoon[7] [29]
  • Temperature under the arm (axillary) is unremarkably under 0.6 °C (1.1 °F) of the core torso temperature.[xxx]

In adults, the normal range of oral temperatures in healthy individuals is 35.seven–37.7 °C (96.3–99.9 °F) amidst men and 33.ii–38.1 °C (91.8–100.half dozen °F) among women, while when taken rectally information technology is 36.7–37.5 °C (98.one–99.5 °F) among men and 36.8–37.i °C (98.2–98.8 °F) among women, and for ear measurement it is 35.v–37.5 °C (95.9–99.5 °F) among men and 35.7–37.five °C (96.3–99.v °F) among women.[31]

Normal body temperatures vary depending on many factors, including age, sex, time of day, ambient temperature, activity level, and more.[32] [33] Normal daily temperature variation has been described as 0.5 °C (0.ix °F).[vii] : 4012 A raised temperature is not always a fever.[32] For example, the temperature rises in healthy people when they exercise, but this is non considered a fever, as the set up signal is normal.[32] On the other paw, a "normal" temperature may be a fever, if information technology is unusually loftier for that person; for example, medically frail elderly people have a decreased power to generate body heat, so a "normal" temperature of 37.3 °C (99.1 °F) may represent a clinically pregnant fever.[32]

Hyperthermia [edit]

Hyperthermia is an top of body temperature over the temperature ready point, due to either too much heat production or not plenty heat loss.[ane] [7] Hyperthermia is thus non considered fever.[7] : 103 Hyperthermia is not to be confused with hyperpyrexia (which is a very high fever).[vii] : 102

Clinically, it is important to distinguish betwixt fever and hyperthermia as hyperthermia may apace lead to decease and does not respond to antipyretic medications. The distinction may however be difficult to make in an emergency setting, and is oftentimes established by identifying possible causes.[7] : 103

Types [edit]

Unlike fever patterns observed in Plasmodium infections.

Diverse patterns of measured patient temperatures accept been observed, some of which may be indicative of a item medical diagnosis:

  • Continuous fever, where temperature remains in a higher place normal throughout the day and does not fluctuate more than 1 °C in 24 hours[34] (e.g. in bacterial pneumonia, typhoid, infective endocarditis, tuberculosis, or typhus[35]);[36]
  • Intermittent fever, where the temperature elevation is present only for a certain menses, later cycling dorsum to normal (due east.m., in malaria, leishmaniasis, pyemia, sepsis,[37] or African trypanosomiasis[38]);
  • Remittent fever, where the temperature remains higher up normal throughout the day and fluctuates more than 1 °C in 24 hours[39] (e.g., in infective endocarditis, or brucellosis[twoscore])
  • Pel–Ebstein fever is a cyclic fever that is rarely seen in patients with Hodgkin's lymphoma.
  • Undulant fever, seen in brucellosis
  • Typhoid fever is an case of continuous fever and it shows a characteristic pace-ladder blueprint, a pace-wise increase in temperature with a high plateau.[41]

Amongst the types of intermittent fever are ones specific to cases of malaria caused by dissimilar pathogens. These are:[42] [43]

  • Quotidian fever, with a 24-hour periodicity, typical of malaria caused by Plasmodium knowlesi (P. knowlesi);[44] [45]
  • Tertian fever, with a 48-hour periodicity, typical of later course malaria caused past P. falciparum, P. vivax, or P. ovale;[42]
  • Quartan fever, with a 72-hour periodicity, typical of later course malaria caused by P. malariae.[42]

In add-on, there is disagreement regarding whether a specific fever pattern is associated with Hodgkin's lymphoma—the Pel–Ebstein fever, with patients argued to present loftier temperature for one week, followed past depression for the next calendar week, and so on, where the generality of this pattern is debated.[46] [ needs update ]

Persistent fever that cannot be explained after repeated routine clinical inquiries is chosen fever of unknown origin.[7] A neutropenic fever, also called febrile neutropenia, is a fever in the absence of normal immune arrangement role.[ commendation needed ] Because of the lack of infection-fighting neutrophils, a bacterial infection can spread chop-chop; this fever is, therefore, usually considered to require urgent medical attending.[47] This kind of fever is more commonly seen in people receiving immune-suppressing chemotherapy than in plain good for you people.[ citation needed ]

Hyperpyrexia [edit]

Hyperpyrexia is an farthermost elevation of body temperature which, depending upon the source, is classified as a core body temperature greater than or equal to 40.0 or 41.0 °C (104.0 or 105.viii °F); the range of hyperpyrexias include cases considered severe (≥ xl °C) and farthermost (≥ 42 °C).[7] [48] [49] It differs from hyperthermia in that one's thermoregulatory system's set indicate for body temperature is set above normal, then estrus is generated to attain it. In dissimilarity, hyperthermia involves body temperature rising in a higher place its set up point due to outside factors.[seven] [50] The loftier temperatures of hyperpyrexia are considered medical emergencies, every bit they may indicate a serious underlying condition or atomic number 82 to astringent morbidity (including permanent brain harm), or to death.[51] A common cause of hyperpyrexia is an intracranial hemorrhage.[7] Other causes in emergency room settings include sepsis, Kawasaki syndrome,[52] neuroleptic malignant syndrome, drug overdose, serotonin syndrome, and thyroid tempest.[51]

Differential diagnosis [edit]

Fever is a common symptom of many medical conditions:

  • Infectious disease, e.grand., COVID-19,[xiii] Dengue, Ebola, gastroenteritis, HIV, influenza, Lyme disease, malaria, mononucleosis, as well as infections of the skin, e.1000., abscesses and boils.[53] [54] [55] [56] [57] [58]
  • Immunological diseases, due east.g., relapsing polychondritis,[59] autoimmune hepatitis, granulomatosis with polyangiitis, Horton affliction, inflammatory bowel diseases, Kawasaki disease, lupus erythematosus, sarcoidosis, and Withal's affliction;[ citation needed ]
  • Tissue destruction, as a issue of cerebral bleeding, beat syndrome, hemolysis, infarction, rhabdomyolysis, surgery, etc.;[ citation needed ]
  • Cancers, particularly blood cancers such as leukemia and lymphomas;[60]
  • Metabolic disorders, e.g., gout, and porphyria;[ citation needed ] and
  • Inherited metabolic disorder, e.g., Fabry affliction.[7]

Adult and pediatric manifestations for the aforementioned disease may differ; for instance, in COVID-19, ane metastudy describes 92.8% of adults versus 43.ix% of children presenting with fever.[xiii]

In addition, fever tin result from a reaction to an incompatible blood product.[61]

Teething is non a cause of fever.[62]

Selective reward [edit]

Hyperthermia: Characterized on the left. Normal trunk temperature (thermoregulatory set betoken) is shown in green, while the hyperthermic temperature is shown in red. As can be seen, hyperthermia tin can be conceptualized as an increment above the thermoregulatory set point.
Hypothermia: Characterized in the center: Normal trunk temperature is shown in green, while the hypothermic temperature is shown in blue. As tin exist seen, hypothermia can exist conceptualized every bit a subtract below the thermoregulatory set betoken.
Fever: Characterized on the correct: Normal body temperature is shown in dark-green. It reads "New Normal" because the thermoregulatory ready point has risen. This has acquired what was the normal body temperature (in blue) to be considered hypothermic.

Scholars viewing fever from an organismal and evolutionary perspective notation the value to an organism of having a fever response, in particular in response to infective disease.[16] [63] [64] On the other hand, while fever evolved as a defense mechanism, studies have not been consistent on whether treating fever by and large worsens or improves mortality risk in a modern clinical setting.[xvi] [17] [65] [66] [67] Benefits or harms may depend on the blazon of infection, health status of the patient & other factors.[16] [65] [66] [67] Studies using warm-blooded vertebrates propose that they recover more rapidly from infections or disquisitional illness due to fever.[68] Other studies suggest reduced mortality in bacterial infections when fever was present.[69] Fever is thought to contribute to host defense,[16] every bit the reproduction of pathogens with strict temperature requirements can exist hindered, and the rates of some of import immunological reactions[ clarification needed ] are increased by temperature.[70] Fever has been described in teaching texts as assisting the healing procedure in various ways, including:

  • increased mobility of leukocytes;[71] : 1044 [ verification needed ]
  • enhanced leukocyte phagocytosis;[71] : 1044 [ verification needed ]
  • decreased endotoxin effects;[71] : 1044 [ verification needed ] and
  • increased proliferation of T cells.[71] : 1044 [ verification needed ] [72] : 212 [ verification needed ]

Pathophysiology [edit]

Hypothalamus [edit]

Temperature is regulated in the hypothalamus. The trigger of a fever, chosen a pyrogen, results in the release of prostaglandin E2 (PGE2). PGE2 in turn acts on the hypothalamus, which creates a systemic response in the torso, causing rut-generating effects to match a new higher temperature ready point. There are four receptors in which PGE2 can bind (EP1-4), with a previous study showing the EP3 subtype is what mediates the fever response.[73] Hence, the hypothalamus can exist seen as working like a thermostat.[7] When the ready point is raised, the torso increases its temperature through both active generation of heat and retention of oestrus. Peripheral vasoconstriction both reduces heat loss through the skin and causes the person to experience cold. Norepinephrine increases thermogenesis in brown adipose tissue, and muscle wrinkle through shivering raises the metabolic charge per unit.[74]

If these measures are insufficient to brand the claret temperature in the brain friction match the new set betoken in the hypothalamus, the brain orchestrates heat effector mechanisms via the autonomic nervous organisation or primary motor eye for shivering. These may be:[ citation needed ]

  • Increased estrus production by increased muscle tone, shivering (musculus movements to produce heat) and release of hormones like epinephrine; and
  • Prevention of estrus loss, east.thousand., through vasoconstriction.

When the hypothalamic set point moves back to baseline—either spontaneously or via medication—normal functions such as sweating, and the reverse of the foregoing processes (eastward.g., vasodilation, end of shivering, and nonshivering oestrus product) are used to absurd the body to the new, lower setting.[ citation needed ]

This contrasts with hyperthermia, in which the normal setting remains, and the body overheats through undesirable retention of excess rut or over-production of heat. Hyperthermia is usually the result of an excessively hot environment (oestrus stroke) or an adverse reaction to drugs. Fever can be differentiated from hyperthermia by the circumstances surrounding it and its response to anti-pyretic medications.[7] [ verification needed ]

In infants, the autonomic nervous organization may also activate brown adipose tissue to produce oestrus (non-practice-associated thermogenesis, also known every bit non-shivering thermogenesis).[ citation needed ]

Increased heart rate and vasoconstriction contribute to increased blood force per unit area in fever.[ commendation needed ]

Pyrogens [edit]

A pyrogen is a substance that induces fever.[75] In the presence of an infectious agent, such every bit bacteria, viruses, viroids, etc., the immune response of the body is to inhibit their growth and eliminate them. The most mutual pyrogens are endotoxins, which are lipopolysaccharides (LPS) produced by Gram-negative leaner such as Due east. coli. But pyrogens include not-endotoxic substances (derived from microorganisms other than gram-negative-bacteria or from chemical substances) as well.[76] The types of pyrogens include internal (endogenous) and external (exogenous) to the body.[ citation needed ]

The "pyrogenicity" of given pyrogens varies: in extreme cases, bacterial pyrogens tin can act as superantigens and crusade rapid and dangerous fevers.[77]

Endogenous [edit]

Endogenous pyrogens are cytokines released from monocytes (which are role of the immune arrangement).[78] In full general, they stimulate chemic responses, often in the presence of an antigen, leading to a fever. Whilst they can exist a product of external factors like exogenous pyrogens, they can also be induced by internal factors like damage associated from molecular patterns such equally cases like rheumatoid arthritis or lupus.[79]

Major endogenous pyrogens are interleukin 1 (α and β)[80] : 1237–1248 and interleukin 6 (IL-6).[81] Minor endogenous pyrogens include interleukin-8, tumor necrosis factor-β, macrophage inflammatory protein-α and macrophage inflammatory protein-β also as interferon-α, interferon-β, and interferon-γ.[80] : 1237–1248 Tumor necrosis factor-α (TNF) also acts equally a pyrogen, mediated by interleukin 1 (IL-1) release.[82] These cytokine factors are released into general circulation, where they migrate to the brain'due south circumventricular organs where they are more easily absorbed than in areas protected by the blood–brain barrier.[ citation needed ] The cytokines then demark to endothelial receptors on vessel walls to receptors on microglial cells, resulting in activation of the arachidonic acrid pathway.[ citation needed ]

Of these, IL-1β, TNF, and IL-six are able to raise the temperature setpoint of an organism and cause fever. These proteins produce a cyclooxygenase which induces the hypothalamic production of PGE2 which then stimulates the release of neurotransmitters such as circadian adenosine monophosphate and increases torso temperature.[83]

Exogenous [edit]

Exogenous pyrogens are external to the body and are of microbial origin. In general, these pyrogens, including bacterial prison cell wall products, may act on Toll-similar receptors in the hypothalamus and elevate the thermoregulatory setpoint.[84]

An example of a class of exogenous pyrogens are bacterial lipopolysaccharides (LPS) present in the cell wall of gram-negative bacteria. According to one machinery of pyrogen action, an immune system protein, lipopolysaccharide-binding protein (LBP), binds to LPS, and the LBP–LPS complex then binds to a CD14 receptor on a macrophage. The LBP-LPS binding to CD14 results in cellular synthesis and release of various endogenous cytokines, e.k., interleukin 1 (IL-ane), interleukin half dozen (IL-6), and tumor necrosis factor-alpha (TNFα). A further downstream event is activation of the arachidonic acid pathway.[85]

PGE2 release [edit]

PGE2 release comes from the arachidonic acid pathway. This pathway (as information technology relates to fever), is mediated by the enzymes phospholipase A2 (PLA2), cyclooxygenase-two (COX-2), and prostaglandin E2 synthase. These enzymes ultimately mediate the synthesis and release of PGE2.[ citation needed ]

PGE2 is the ultimate mediator of the delirious response. The setpoint temperature of the body will remain elevated until PGE2 is no longer present. PGE2 acts on neurons in the preoptic expanse (POA) through the prostaglandin E receptor three (EP3). EP3-expressing neurons in the POA innervate the dorsomedial hypothalamus (DMH), the rostral raphe pallidus nucleus in the medulla oblongata (rRPa), and the paraventricular nucleus (PVN) of the hypothalamus. Fever signals sent to the DMH and rRPa lead to stimulation of the sympathetic output arrangement, which evokes non-shivering thermogenesis to produce torso estrus and pare vasoconstriction to subtract oestrus loss from the torso surface. It is presumed that the innervation from the POA to the PVN mediates the neuroendocrine effects of fever through the pathway involving pituitary gland and various endocrine organs.[ citation needed ]

Management [edit]

Fever does not necessarily need to be treated,[86] and nigh people with a fever recover without specific medical attention.[87] Although it is unpleasant, fever rarely rises to a dangerous level fifty-fifty if untreated. Damage to the brain generally does not occur until temperatures accomplish 42 °C (107.6 °F), and it is rare for an untreated fever to exceed twoscore.six °C (105 °F).[88] Treating fever in people with sepsis does not affect outcomes.[89]

Conservative measures [edit]

Limited show supports sponging or bathing feverish children with tepid water.[90] The use of a fan or air workout may somewhat reduce the temperature and increase comfort. If the temperature reaches the extremely loftier level of hyperpyrexia, aggressive cooling is required (by and large produced mechanically via conduction by applying numerous ice packs across most of the body or direct submersion in water ice h2o).[51] In general, people are advised to keep fairly hydrated.[91] Whether increased fluid intake improves symptoms or shortens respiratory illnesses such equally the mutual cold is non known.[92]

Medications [edit]

Medications that lower fevers are called antipyretics. The antipyretic ibuprofen is effective in reducing fevers in children.[93] It is more effective than acetaminophen (paracetamol) in children.[93] Ibuprofen and acetaminophen may be safely used together in children with fevers.[94] [95] The efficacy of acetaminophen by itself in children with fevers has been questioned.[96] Ibuprofen is besides superior to aspirin in children with fevers.[97] Additionally, aspirin is not recommended in children and young adults (those under the age of 16 or 19 depending on the country) due to the chance of Reye's syndrome.[98]

Using both paracetamol and ibuprofen at the aforementioned time or alternate between the two is more effective at decreasing fever than using merely paracetamol or ibuprofen.[99] It is not clear if information technology increases child comfort.[99] Response or nonresponse to medications does not predict whether or not a child has a serious disease.[100]

With respect to the effect of antipyretics on the risk of decease in those with infection, studies have found mixed results equally of 2019.[101] Animal models have found worsened outcomes with the use of antipyretics in influenza equally of 2010 but they have not been studied for this apply in humans.[102]

Epidemiology [edit]

Fever is one of the most common medical signs.[ii] It is office of about 30% of healthcare visits by children,[2] and occurs in upward to 75% of adults who are seriously sick.[11] About 5% of people who go to an emergency room have a fever.[103]

History [edit]

A number of types of fever were known equally early as 460 BC to 370 BC when Hippocrates was practicing medicine including that due to malaria (tertian or every 2 days and quartan or every iii days).[104] Information technology also became clear around this time that fever was a symptom of disease rather than a illness in and of itself.[104]

Infections presenting with fever were a major source of bloodshed in humans for about 200,000 years. Until the late nineteenth century, approximately half of all humans died from infections before the age of fifteen.[105]

An older term, febricula (a diminutive form of the Latin word for fever), was once used to refer to a low-course fever lasting only a few days. This term fell out of use in the early on 20th century, and the symptoms it referred to are now thought to have been caused mainly past various minor viral respiratory infections.[106]

Society and civilisation [edit]

Fever is often viewed with greater concern by parents and healthcare professionals than might be deserved, a miracle known as fever phobia,[two] [107] which is based in both caregiver's and parents' misconceptions about fever in children. Amongst them, many parents incorrectly believe that fever is a illness rather than a medical sign, that even low fevers are harmful, and that any temperature even briefly or slightly above the oversimplified "normal" number marked on a thermometer is a clinically pregnant fever.[107] They are likewise afraid of harmless side effects like febrile seizures and dramatically overestimate the likelihood of permanent damage from typical fevers.[107] The underlying problem, co-ordinate to professor of pediatrics Barton D. Schmitt, is that "equally parents we tend to suspect that our children'south brains may cook."[108] As a result of these misconceptions parents are broken-hearted, requite the kid fever-reducing medicine when the temperature is technically normal or simply slightly elevated, and interfere with the kid's sleep to give the child more medicine.[107]

Other species [edit]

Fever is an important characteristic for the diagnosis of disease in domestic animals. The trunk temperature of animals, which is taken rectally, is different from one species to another. For example, a horse is said to have a fever above 101 °F ( 38.3 °C).[109] In species that allow the torso to accept a wide range of "normal" temperatures, such as camels,[110] whose body temperature varies as the environmental temperature varies,[111] the trunk temperature which constitutes a febrile state differs depending on the ecology temperature.[112] Fever can also exist behaviorally induced by invertebrates that do not have allowed-system based fever. For instance, some species of grasshopper will thermoregulate to achieve body temperatures that are 2–5 °C higher than normal in order to inhibit the growth of fungal pathogens such every bit Beauveria bassiana and Metarhizium acridum.[113] Honeybee colonies are too able to induce a fever in response to a fungal parasite Ascosphaera apis.[113]

References [edit]

  1. ^ a b c d eastward f thousand h i Axelrod YK, Diringer MN (May 2008). "Temperature management in acute neurologic disorders". Neurologic Clinics. 26 (two): 585–603, xi. doi:10.1016/j.ncl.2008.02.005. PMID 18514828.
  2. ^ a b c d e f grand h i j one thousand fifty Sullivan JE, Farrar HC (March 2011). "Fever and antipyretic use in children". Pediatrics. 127 (3): 580–87. doi:10.1542/peds.2010-3852. PMID 21357332.
  3. ^ a b c Huether, Sue E. (2014). Pathophysiology: The Biologic Basis for Disease in Adults and Children (7th ed.). Elsevier Health Sciences. p. 498. ISBN978-0323293754.
  4. ^ a b CDC Staff (31 March 2020). "Taking Care of Someone Who is Ill: Caring for Someone Sick at Domicile". Archived from the original on 24 March 2015. Retrieved 8 May 2015.
  5. ^ a b Kluger MJ (2015). Fever: Its Biology, Evolution, and Function. Princeton University Press. p. 57. ISBN978-1400869831.
  6. ^ a b c Garmel GM, Mahadevan SV, eds. (2012). "Fever in adults". An introduction to clinical emergency medicine (2nd ed.). Cambridge: Cambridge University Press. p. 375. ISBN978-0521747769.
  7. ^ a b c d eastward f yard h i j k l m n o p Dinarello CA, Porat R (2018). "Chapter 15: Fever". In Jameson JL, Fauci Every bit, Kasper DL, Hauser SL, Longo DL, Loscalzo, J (eds.). Harrison'southward Principles of Internal Medicine. Vol. 1–ii (20th ed.). New York, NY: McGraw-Hill. ISBN9781259644030 . Retrieved 31 March 2020.
  8. ^ a b c d e f Laupland KB (July 2009). "Fever in the critically ill medical patient". Critical Care Medicine. 37 (seven Suppl): S273-8. doi:ten.1097/CCM.0b013e3181aa6117. PMID 19535958.
  9. ^ a b c d eastward
  10. ^ a b Garmel GM, Mahadevan SV, eds. (2012). An introduction to clinical emergency medicine (2nd ed.). Cambridge: Cambridge University Press. p. 401. ISBN978-0521747769.
  11. ^ a b c Kiekkas P, Aretha D, Bakalis Northward, Karpouhtsi I, Marneras C, Baltopoulos GI (August 2013). "Fever effects and handling in critical care: literature review". Australian Critical Care. 26 (three): 130–35. doi:10.1016/j.aucc.2012.10.004. PMID 23199670.
  12. ^ a b c Garmel GM, Mahadevan SV, eds. (2012). An introduction to clinical emergency medicine (2nd ed.). Cambridge: Cambridge Academy Press. p. five. ISBN978-0521747769.
  13. ^ a b c Rodriguez-Morales AJ, Cardona-Ospina JA, Gutiérrez-Ocampo E, Villamizar-Peña R, Holguin-Rivera Y, Escalera-Antezana JP, Alvarado-Arnez LE, Bonilla-Aldana DK, Franco-Paredes C (thirteen March 2020). "Clinical, laboratory and imaging features of COVID-19: A systematic review and meta-analysis". Travel Medicine and Infectious disease. 34: 101623. doi:ten.1016/j.tmaid.2020.101623. PMC7102608. PMID 32179124.
  14. ^ Dayal R, Agarwal D (Jan 2016). "Fever in Children and Fever of Unknown Origin". Indian Journal of Pediatrics. 83 (1): 38–43. doi:x.1007/s12098-015-1724-iv. PMID 25724501. S2CID 34481402.
  15. ^ "Fever". MedlinePlus. 30 August 2014. Archived from the original on 11 May 2009.
  16. ^ a b c d e Schaffner A (March 2006). "Fieber – nützliches oder schädliches, zu behandelndes Symptom?" [Fever–useful or baneful symptom that should be treated?]. Therapeutische Umschau (in German). 63 (three): 185–88. doi:x.1024/0040-5930.63.3.185. PMID 16613288. Abstruse alone is in German language and in English.
  17. ^ a b Niven DJ, Stelfox HT, Laupland KB (June 2013). "Antipyretic therapy in febrile critically sick adults: A systematic review and meta-analysis". Periodical of Disquisitional Care. 28 (3): 303–10. doi:ten.1016/j.jcrc.2012.09.009. PMID 23159136.
  18. ^ Crocetti M, Moghbeli N, Serwint J (June 2001). "Fever Phobia Revisited: Have Parental Misconceptions About Fever Changed in 20 Years?". Pediatrics. 107 (six): 1241–1246. doi:10.1542/peds.107.6.1241. PMID 11389237.
  19. ^ Kelley KW, Bluthé RM, Dantzer R, Zhou JH, Shen WH, Johnson RW, Broussard SR (February 2003). "Cytokine-induced sickness behavior". Brain, Beliefs, and Amnesty. 17 Suppl 1 (1): S112–18. doi:10.1016/S0889-1591(02)00077-half dozen. PMID 12615196. S2CID 25400611.
  20. ^ Adamis D, Treloar A, Martin FC, Macdonald AJ (December 2007). "A brief review of the history of delirium every bit a mental disorder". History of Psychiatry. 18 (72 Pt 4): 459–69. doi:ten.1177/0957154X07076467. PMID 18590023. S2CID 24424207.
  21. ^ Marx J (2006). Rosen'southward emergency medicine : concepts and clinical practice (sixth ed.). Philadelphia: Mosby/Elsevier. p. 2239. ISBN978-0-323-02845-5. OCLC 58533794.
  22. ^ Hutchison JS, Ward RE, Lacroix J, Hébert PC, Barnes MA, Bohn DJ, et al. (June 2008). "Hypothermia therapy afterwards traumatic encephalon injury in children". The New England Journal of Medicine. 358 (23): 2447–56. doi:10.1056/NEJMoa0706930. PMID 18525042.
  23. ^ Pryor JA, Prasad Equally (2008). Physiotherapy for Respiratory and Cardiac Problems: Adults and Paediatrics. Elsevier Health Sciences. p. 8. ISBN978-0702039744. Body temperature is maintained within the range 36.5-37.5 °C. It is lowest in the early on morning and highest in the afternoon.
  24. ^ Grunau BE, Wiens MO, Brubacher JR (September 2010). "Dantrolene in the treatment of MDMA-related hyperpyrexia: a systematic review". Cjem. 12 (5): 435–42. doi:10.1017/s1481803500012598. PMID 20880437. Dantrolene may also be associated with improved survival and reduced complications, peculiarly in patients with extreme (≥ 42 °C) or astringent (≥ 40 °C) hyperpyrexia
  25. ^ Sharma HS, ed. (2007). Neurobiology of Hyperthermia (1st ed.). Elsevier. pp. 175–177, 485. ISBN9780080549996 . Retrieved 19 November 2016. Despite the myriad of complications associated with heat illness, an tiptop of core temperature above 41.0 °C (often referred to as fever or hyperpyrexia) is the most widely recognized symptom of this syndrome.
  26. ^ Niven DJ, Gaudet JE, Laupland KB, Mrklas KJ, Roberts DJ, Stelfox HT (November 2015). "Accuracy of peripheral thermometers for estimating temperature: a systematic review and meta-analysis". Register of Internal Medicine. 163 (ten): 768–77. doi:10.7326/M15-1150. PMID 26571241. S2CID 4004360.
  27. ^ "Measuring a Baby's Temperature". world wide web.hopkinsmedicine.org. Archived from the original on 3 November 2019. Retrieved 10 September 2019.
  28. ^ "Tips for taking your child's temperature". Mayo Clinic . Retrieved 10 September 2019.
  29. ^ Barone JE (August 2009). "Fever: Fact and fiction". The Periodical of Trauma. 67 (2): 406–09. doi:10.1097/TA.0b013e3181a5f335. PMID 19667898.
  30. ^ Pecoraro, Valentina; Petri, Davide; Costantino, Giorgio; Squizzato, Alessandro; Moja, Lorenzo; Virgili, Gianni; Lucenteforte, Ersilia (25 November 2020). "The diagnostic accuracy of digital, infrared and mercury-in-glass thermometers in measuring body temperature: a systematic review and network meta-analysis". Internal and Emergency Medicine. Springer Scientific discipline and Business concern Media LLC. sixteen (4): 1071–1083. doi:10.1007/s11739-020-02556-0. ISSN 1828-0447. PMC7686821. PMID 33237494.
  31. ^ Sund-Levander M, Forsberg C, Wahren LK (June 2002). "Normal oral, rectal, tympanic and axillary body temperature in adult men and women: a systematic literature review". Scandinavian Journal of Caring Sciences. xvi (two): 122–28. doi:10.1046/j.1471-6712.2002.00069.ten. PMID 12000664.
  32. ^ a b c d Garami, András; Székely, Miklós (6 May 2014). "Trunk temperature". Temperature: Multidisciplinary Biomedical Journal. one (ane): 28–29. doi:10.4161/temp.29060. ISSN 2332-8940. PMC4972507. PMID 27583277.
  33. ^ "Body temperature: What is the new normal?". www.medicalnewstoday.com. 12 Jan 2020. Retrieved vii April 2020.
  34. ^ Ogoina D (August 2011). "Fever, fever patterns and diseases called 'fever' – a review". Journal of Infection and Public Health. 4 (3): 108–24. doi:ten.1016/j.jiph.2011.05.002. PMID 21843857.
  35. ^ Typhoid fever may show a specific fever blueprint, a Wunderlich curve, with a slow stepwise increase and a loftier plateau (drops due to fever-reducing drugs are excluded).[ citation needed ]
  36. ^ Dall, Lawrence; Stanford, James F. (1990). "Fever, Chills, and Night Sweats". In Walker, H. Kenneth; Hall, Westward. Dallas; Hurst, J. Willis (eds.). Clinical Methods: The History, Physical, and Laboratory Examinations (3rd ed.). Boston: Butterworths. ISBN0-409-90077-X. PMID 21250166.
  37. ^ Inayatullah, Muhammad; Nasir, Shabbir Ahmed (2016). Bedside Techniques: Methods of Clinical Examination (fourth ed.). ISBN978-969-494-920-8. [ page needed ]
  38. ^ "CDC - African Trypanosomiasis - Disease". www.cdc.gov. 28 April 2020. Retrieved 18 July 2021.
  39. ^ The Medical News and Library. Lea & Blanchard. 1843. p. 96 – via Internet Archive. remittent fever news.
  40. ^ "Brucella/Brucellosis". The Lecturio Medical Concept Library . Retrieved 19 July 2021.
  41. ^ "Enteric Fever (Typhoid Fever)". The Lecturio Medical Concept Library. 27 Baronial 2020. Retrieved 19 July 2021.
  42. ^ a b c Ferri FF (2009). "Affiliate 332. Protozoal infections". Ferri's Color Atlas and Text of Clinical Medicine. Elsevier Health Sciences. pp. 1159ff. ISBN9781416049197. Archived from the original on iii June 2016. Retrieved 31 March 2020.
  43. ^ Muhammad I, Nasir, SA (2009). Bedside Techniques: Methods of Clinical Test. Multan, Pakistan: Saira Publishers/Salamat Iqbal Printing. [ page needed ] [ better source needed ]
  44. ^ Singh, B.; Daneshvar, C. (1 Apr 2013). "Human Infections and Detection of Plasmodium knowlesi". Clinical Microbiology Reviews. 26 (two): 165–184. doi:10.1128/CMR.00079-12. PMC3623376. PMID 23554413.
  45. ^ Chin, W.; Contacos, P. Grand.; Coatney, G. R.; Kimball, H. R. (twenty August 1965). "A Naturally Caused Quotidian-Type Malaria in Man Transferable to Monkeys". Scientific discipline. 149 (3686): 865. Bibcode:1965Sci...149..865C. doi:10.1126/science.149.3686.865. PMID 14332847. S2CID 27841173.
  46. ^ Hilson AJ (July 1995). "Pel-Ebstein fever". The New England Journal of Medicine. 333 (ane): 66–67. doi:10.1056/NEJM199507063330118. PMID 7777006. , which cites Richard Asher'south lecture, "Making Sense" [Lancet (1959) 2: 359].
  47. ^ White, Lindsey; Ybarra, Michael (1 Dec 2017). "Neutropenic Fever". Hematology/Oncology Clinics of N America. 31 (6): 981–993. doi:10.1016/j.hoc.2017.08.004. PMID 29078933 – via ClinicalKey.
  48. ^ Grunau BE, Wiens MO, Brubacher JR (September 2010). "Dantrolene in the handling of MDMA-related hyperpyrexia: a systematic review". Canadian Journal of Emergency Medicine. 12 (5): 435–42. doi:10.1017/s1481803500012598. PMID 20880437. Dantrolene may also be associated with improved survival and reduced complications, peculiarly in patients with extreme (≥ 42 °C) or astringent (≥ 40 °C) hyperpyrexia
  49. ^ Sharma HS, ed. (2007). Neurobiology of Hyperthermia (1st ed.). Elsevier. pp. 175–77, 485. ISBN978-0080549996. Archived from the original on 8 September 2017. Retrieved 19 Nov 2016. Despite the myriad of complications associated with heat illness, an elevation of core temperature above 41.0 °C (often referred to as fever or hyperpyrexia) is the most widely recognized symptom of this syndrome.
  50. ^ Run into section in Chapter fifteen therein, the section on "Fever versus hyperthermia".
  51. ^ a b c McGugan EA (March 2001). "Hyperpyrexia in the emergency department". Emergency Medicine. 13 (1): 116–xx. doi:10.1046/j.1442-2026.2001.00189.ten. PMID 11476402.
  52. ^ Marx (2006), p. 2506.
  53. ^ Raoult, Didier; Levy, Pierre-Yves; Dupont, Hervé Tissot; Chicheportiche, Colette; Tamalet, Catherine; Gastaut, Jean-Albert; Salducci, Jacques (Jan 1993). "Q fever and HIV infection". AIDS. 7 (1): 81–86. doi:10.1097/00002030-199301000-00012. ISSN 0269-9370. PMID 8442921.
  54. ^ French, Neil; Nakiyingi, Jessica; Lugada, Eric; Watera, Christine; Whitworth, James A. 1000.; Gilks, Charles F. (May 2001). "Increasing rates of malarial fever with deteriorating immune condition in HIV-1-infected Ugandan adults". AIDS. 15 (7): 899–906. doi:10.1097/00002030-200105040-00010. ISSN 0269-9370. PMID 11399962. S2CID 25470703. Archived from the original on 22 February 2022.
  55. ^ Heymann, D. L.; Weisfeld, J. Due south.; Webb, P. A.; Johnson, Thousand. M.; Cairns, T.; Berquist, H. (one September 1980). "Ebola Hemorrhagic Fever: Tandala, Zaire, 1977-1978". Journal of Infectious Diseases. 142 (three): 372–376. doi:10.1093/infdis/142.3.372. ISSN 0022-1899. PMID 7441008.
  56. ^ Feldmann, Heinz; Geisbert, Thomas Due west (March 2011). "Ebola haemorrhagic fever". The Lancet. 377 (9768): 849–862. doi:10.1016/s0140-6736(10)60667-8. ISSN 0140-6736. PMC3406178. PMID 21084112.
  57. ^ Oakley, Miranda South.; Gerald, Noel; McCutchan, Thomas F.; Aravind, L.; Kumar, Sanjai (October 2011). "Clinical and molecular aspects of malaria fever". Trends in Parasitology. 27 (10): 442–449. doi:x.1016/j.pt.2011.06.004. ISSN 1471-4922. PMID 21795115.
  58. ^ Colunga-Salas, Pablo; Sánchez-Montes, Sokani; Volkow, Patricia; Ruíz-Remigio, Adriana; Becker, Ingeborg (17 September 2020). "Lyme disease and relapsing fever in Mexico: An overview of human and wildlife infections". PLOS Ane. 15 (9): e0238496. Bibcode:2020PLoSO..1538496C. doi:10.1371/journal.pone.0238496. ISSN 1932-6203. PMC7497999. PMID 32941463.
  59. ^ Puéchal 10, Terrier B, Mouthon L, Costedoat-Chalumeau N, Guillevin L, Le Jeunne C (March 2014). "Relapsing polychondritis". Articulation, Bone, Spine. 81 (2): 118–24. doi:ten.1016/j.jbspin.2014.01.001. PMID 24556284.
  60. ^ "Signs and Symptoms of Cancer | Do I Have Cancer?". www.cancer.org . Retrieved 20 June 2020.
  61. ^ Dean, Laura (2005). Blood transfusions and the immune system. National Center for Biotechnology Information (U.s.).
  62. ^ Massignan C, Cardoso One thousand, Porporatti AL, Aydinoz S, Canto G, Mezzomo LA, Bolan M (March 2016). "Signs and Symptoms of Chief Tooth Eruption: A Meta-analysis". Pediatrics. 137 (3): e20153501. doi:10.1542/peds.2015-3501. PMID 26908659. Archived from the original on 21 February 2016.
  63. ^ SoszyÅ„ski D (2003). "[The pathogenesis and the adaptive value of fever]". Postepy Higieny I Medycyny Doswiadczalnej (in Polish). 57 (v): 531–54. PMID 14737969.
  64. ^ Kluger MJ, Kozak W, Conn CA, Leon LR, Soszynski D (September 1998). "Role of fever in affliction". Register of the New York University of Sciences. 856 (1): 224–33. Bibcode:1998NYASA.856..224K. doi:x.1111/j.1749-6632.1998.tb08329.x. PMID 9917881. S2CID 12408561.
  65. ^ a b Schulman, Carl I.; Namias, Nicholas; Doherty, James; Manning, Ronald J.; Li, Pamela; Elhaddad, Ahmed; Lasko, David; Amortegui, Jose; Dy, Christopher J.; Dlugasch, Lucie; Baracco, Gio; Cohn, Stephen Chiliad. (2005). "The Effect of Antipyretic Therapy upon Outcomes in Critically Ill Patients: A Randomized, Prospective Study". Surgical Infections. 6 (four): 369–375. doi:ten.1089/sur.2005.half dozen.369. PMID 16433601.
  66. ^ a b Drewry, Anne K.; Ablordeppey, Enyo A.; Murray, Ellen T.; Stoll, Carolyn R. T.; Izadi, Sonya R.; Dalton, Catherine Thou.; Hardi, Angela C.; Fowler, Susan A.; Fuller, Brian M.; Colditz, Graham A. (2017). "Antipyretic Therapy in Critically Sick Septic Patients". Critical Care Medicine. 45 (5): 806–813. doi:10.1097/CCM.0000000000002285. PMC5389594. PMID 28221185.
  67. ^ a b https://www.australiancriticalcare.com/commodity/S1036-7314(19)30093-1/fulltext
  68. ^ Su F, Nguyen ND, Wang Z, Cai Y, Rogiers P, Vincent JL (June 2005). "Fever control in septic shock: beneficial or harmful?". Shock. 23 (6): 516–20. PMID 15897803.
  69. ^ Rantala South, Vuopio-Varkila J, Vuento R, Huhtala H, Syrjänen J (April 2009). "Predictors of bloodshed in beta-hemolytic streptococcal bacteremia: a population-based study". The Journal of Infection. 58 (4): 266–72. doi:10.1016/j.jinf.2009.01.015. PMID 19261333.
  70. ^ Fischler MP, Reinhart WH (May 1997). "[Fever: friend or enemy?]". Schweizerische Medizinische Wochenschrift (in German). 127 (20): 864–lxx. PMID 9289813.
  71. ^ a b c d Chicken RF, Hirnle CJ (2003). Fundamentals of Nursing: Human Health and Function (4th ed.). Philadelphia, PA: Lippincott Williams & Wilkins. ISBN9780781758185 . Retrieved 2 Apr 2020. {{cite volume}}: CS1 maint: uses authors parameter (link)
  72. ^ Lewis SM, Dirksen SR, Heitkemper MM (2005). Medical-Surgical Nursing: Cess and Direction of Clinical Problems (6th ed.). Amsterdam, NL: Elsevier-Wellness Sciences. ISBN9780323031059 . Retrieved 2 April 2020. {{cite book}}: CS1 maint: uses authors parameter (link)
  73. ^ Ushikubi F, et al. (September 1998). "Impaired delirious response in mice lacking the prostaglandin Due east receptor subtype EP3". Nature. 395 (6699): 281–284. Bibcode:1998Natur.395..281U. doi:ten.1038/26233. PMID 9751056. S2CID 4420632.
  74. ^ Evans SS, Repasky EA, Fisher DT (June 2015). "Fever and the thermal regulation of amnesty: the immune system feels the oestrus". Nature Reviews. Immunology. 15 (6): 335–49. doi:10.1038/nri3843. PMC4786079. PMID 25976513.
  75. ^ Hagel L, Jagschies Chiliad, Sofer M (ane Jan 2008). "5 - Analysis". Handbook of Procedure Chromatography (second ed.). Bookish Press. pp. 127–145. doi:ten.1016/b978-012374023-6.50007-v. ISBN978-0-12-374023-half dozen.
  76. ^ Kojima Grand (1 Jan 2012). "17 - Biological evaluation and regulation of medical devices in Nippon". In Boutrand J (ed.). Biocompatibility and Performance of Medical Devices . Woodhead Publishing Series in Biomaterials. Woodhead Publishing. pp. 404–448. doi:10.1533/9780857096456.4.404. ISBN978-0-85709-070-half-dozen.
  77. ^ Affairs, Office of Regulatory (3 November 2018). "Pyrogens, Still a Danger". FDA.
  78. ^ Constable PD, Hinchcliff KW, Done SH, Grünberg W, eds. (ane Jan 2017). "four - General Systemic States". Veterinarian Medicine (11th ed.). Westward.B. Saunders. pp. 43–112. doi:ten.1016/b978-0-7020-5246-0.00004-eight. ISBN978-0-7020-5246-0. S2CID 214758182.
  79. ^ Dinarello CA (31 March 2015). "The history of fever, leukocytic pyrogen and interleukin-1". Temperature. two (1): 8–16. doi:x.1080/23328940.2015.1017086. PMC4843879. PMID 27226996.
  80. ^ a b Stitt, John (2008). "Chapter 59: Regulation of Torso Temperature". In Boron WF, Boulpaep, EL (eds.). Medical Physiology: A Cellular and Molecular Approach (2d ed.). Philadelphia, PA: Elsevier Saunders. ISBN9781416031154 . Retrieved 2 Apr 2020.
  81. ^ Murphy, Kenneth (Kenneth M.) (2017). Janeway'due south immunobiology. Weaver, Casey (ninth ed.). New York, NY, USA. pp. 118–119. ISBN978-0-8153-4505-3. OCLC 933586700.
  82. ^ Stefferl A, Hopkins SJ, Rothwell NJ, Luheshi GN (August 1996). "The role of TNF-alpha in fever: opposing deportment of human being and murine TNF-blastoff and interactions with IL-beta in the rat". British Journal of Pharmacology. 118 (8): 1919–24. doi:ten.1111/j.1476-5381.1996.tb15625.x. PMC1909906. PMID 8864524.
  83. ^ Srinivasan 50, Harris MC, Kilpatrick LE (one January 2017). "128 - Cytokines and Inflammatory Response in the Fetus and Neonate". In Polin RA, Abman SH, Rowitch DH, Benitz We (eds.). Fetal and Neonatal Physiology (5th ed.). Elsevier. pp. 1241–1254.e4. doi:10.1016/b978-0-323-35214-7.00128-one. ISBN978-0-323-35214-7.
  84. ^ Wilson ME, Boggild AK (1 Jan 2011). "130 - Fever and Systemic Symptoms". In Guerrant RL, Walker DH, Weller PF (eds.). Tropical Infectious Diseases: Principles, Pathogens and Practice (third ed.). W.B. Saunders. pp. 925–938. doi:ten.1016/b978-0-7020-3935-5.00130-0. ISBN978-0-7020-3935-v.
  85. ^ Roth J, Blatteis CM (October 2014). "Mechanisms of fever production and lysis: Lessons from experimental LPS fever". Comprehensive Physiology. 4 (four): 1563–604. doi:10.1002/cphy.c130033. ISBN9780470650714. PMID 25428854.
  86. ^ Ludwig J, McWhinnie H (May 2019). "Antipyretic drugs in patients with fever and infection: literature review". Br J Nurs. 28 (10): 610–618. doi:10.12968/bjon.2019.28.10.610. PMID 31116598. S2CID 162182092.
  87. ^ "What To Do If You lot Become Sick: 2009 H1N1 and Seasonal Flu". Centers for Disease Control and Prevention. vii May 2009. Archived from the original on three November 2009. Retrieved 1 November 2009.
  88. ^ Edward James Walter and Mike Carraretto (2016). "The neurological and cognitive consequences of hyperthermia". Critical Care. twenty (one): 199. doi:x.1186/s13054-016-1376-four. PMC4944502. PMID 27411704. {{cite journal}}: CS1 maint: uses authors parameter (link)
  89. ^ Drewry AM, Ablordeppey EA, Murray ET, Stoll CR, Izadi SR, Dalton CM, Hardi Air-conditioning, Fowler SA, Fuller BM, Colditz GA (May 2017). "Antipyretic Therapy in Critically Sick Septic Patients: A Systematic Review and Meta-Analysis". Critical Care Medicine. 45 (five): 806–xiii. doi:x.1097/CCM.0000000000002285. PMC5389594. PMID 28221185.
  90. ^ Meremikwu M, Oyo-Ita A (2003). Meremikwu MM (ed.). "Physical methods for treating fever in children". The Cochrane Database of Systematic Reviews (2): CD004264. doi:10.1002/14651858.CD004264. PMC6532675. PMID 12804512.
  91. ^ "Fever". National Institute of Health. Archived from the original on thirty April 2016.
  92. ^ Guppy MP, Mickan SM, Del Mar CB, Thorning S, Rack A (February 2011). "Advising patients to increment fluid intake for treating acute respiratory infections". The Cochrane Database of Systematic Reviews (2): CD004419. doi:10.1002/14651858.CD004419.pub3. PMC7197045. PMID 21328268.
  93. ^ a b Perrott DA, Piira T, Goodenough B, Champion GD (June 2004). "Efficacy and safety of acetaminophen vs ibuprofen for treating children's pain or fever: a meta-analysis". Archives of Pediatrics & Adolescent Medicine. 158 (vi): 521–26. doi:ten.1001/archpedi.158.half-dozen.521. PMID 15184213.
  94. ^ Hay AD, Redmond NM, Costelloe C, Montgomery AA, Fletcher Thousand, Hollinghurst S, Peters TJ (May 2009). "Paracetamol and ibuprofen for the treatment of fever in children: the PITCH randomised controlled trial". Wellness Technology Assessment. 13 (27): iii–iv, nine–x, 1–163. doi:10.3310/hta13270. PMID 19454182.
  95. ^ Southey ER, Soares-Weiser K, Kleijnen J (September 2009). "Systematic review and meta-analysis of the clinical rubber and tolerability of ibuprofen compared with paracetamol in paediatric hurting and fever". Current Medical Enquiry and Stance. 25 (ix): 2207–22. doi:10.1185/03007990903116255. PMID 19606950. S2CID 31653539.
  96. ^ Meremikwu M, Oyo-Ita A (2002). "Paracetamol for treating fever in children". The Cochrane Database of Systematic Reviews (2): CD003676. doi:x.1002/14651858.CD003676. PMC6532671. PMID 12076499.
  97. ^ Autret E, Reboul-Marty J, Henry-Launois B, Laborde C, Courcier S, Goehrs JM, Languillat One thousand, Launois R (1997). "Evaluation of ibuprofen versus aspirin and paracetamol on efficacy and condolement in children with fever". European Journal of Clinical Pharmacology. 51 (5): 367–71. doi:10.1007/s002280050215. PMID 9049576. S2CID 27519225.
  98. ^ "2.9 Antiplatelet drugs". British National Formulary for Children. British Medical Clan and Royal Pharmaceutical Society of Neat Britain. 2007. p. 151.
  99. ^ a b Wong T, Stang AS, Ganshorn H, Hartling L, Maconochie IK, Thomsen AM, Johnson DW (October 2013). "Combined and alternating paracetamol and ibuprofen therapy for febrile children". The Cochrane Database of Systematic Reviews (ten): CD009572. doi:10.1002/14651858.CD009572.pub2. PMC6532735. PMID 24174375.
  100. ^ Male monarch D (August 2013). "Question 2: does a failure to respond to antipyretics predict serious illness in children with a fever?". Archives of Disease in Childhood. 98 (8): 644–46. doi:ten.1136/archdischild-2013-304497. PMID 23846358. S2CID 32438262.
  101. ^ Ludwig J, McWhinnie H (May 2019). "Antipyretic drugs in patients with fever and infection: literature review". British Journal of Nursing. 28 (ten): 610–618. doi:ten.12968/bjon.2019.28.10.610. PMID 31116598. S2CID 162182092.
  102. ^ Eyers Due south, Weatherall M, Shirtcliffe P, Perrin One thousand, Beasley R (Oct 2010). "The event on bloodshed of antipyretics in the treatment of influenza infection: systematic review and meta-analysis". Journal of the Regal Society of Medicine. 103 (ten): 403–11. doi:ten.1258/jrsm.2010.090441. PMC2951171. PMID 20929891.
  103. ^ Nassisi D, Oishi ML (January 2012). "Evidence-based guidelines for evaluation and antimicrobial therapy for common emergency department infections". Emergency Medicine Practice. xiv (i): ane–28, quiz 28–29. PMID 22292348.
  104. ^ a b Sajadi MM, Bonabi R, Sajadi MR, Mackowiak PA (October 2012). "Akhawayni and the first fever bend". Clinical Infectious Diseases. 55 (7): 976–80. doi:ten.1093/cid/cis596. PMID 22820543.
  105. ^ Casanova, Jean-Laurent; Abel, Laurent (2021). "Lethal Infectious Diseases every bit Inborn Errors of Amnesty: Toward a Synthesis of the Germ and Genetic Theories". Almanac Review of Pathology: Mechanisms of Disease. 16: 23–l. doi:x.1146/annurev-pathol-031920-101429. PMC7923385. PMID 32289233.
  106. ^ Straus, Bernard (September 1970) [first presented at a conference on 27 May 1970]. "Defunct and Dying Diseases" (PDF). Bulletin of the New York University of Medicine. 46 (9): 686–706. PMC1749762. PMID 4916301. Archived from the original on 6 February 2020.
  107. ^ a b c d Crocetti Thou, Moghbeli N, Serwint J (June 2001). "Fever Phobia Revisited: Have Parental Misconceptions About Fever Changed in 20 Years?". Pediatrics. 107 (6): 1241–1246. doi:10.1542/peds.107.6.1241. PMID 11389237. Retrieved 31 March 2020.
  108. ^ Klass, Perri (ten January 2011). "Lifting a Veil of Fright to Meet a Few Benefits of Fever". The New York Times. Archived from the original on 29 September 2015.
  109. ^ "Equusite Vital Signs". equusite.com. Archived from the original on 26 March 2010. Retrieved 22 March 2010.
  110. ^ Schmidt-Nielsen Thou, Schmidt-Nielsen B, Jarnum SA, Houpt TR (Jan 1957). "Body temperature of the camel and its relation to water economy". The American Journal of Physiology. 188 (1): 103–12. doi:10.1152/ajplegacy.1956.188.one.103. PMID 13402948.
  111. ^ Leese A (March 1917). ""Tips" on camels, for veterinary surgeons on active service". The British Veterinary Periodical. 73: 81 – via Google Books.
  112. ^ Tefera M (July 2004). "Observations on the clinical examination of the camel (Camelus dromedarius) in the field". Tropical Beast Health and Production. 36 (five): 435–49. doi:10.1023/b:trop.0000035006.37928.cf. PMID 15449833. S2CID 26358556.
  113. ^ a b Thomas MB, Blanford S (July 2003). "Thermal biology in insect-parasite interactions". Trends in Ecology & Evolution. 18 (7): 344–l. doi:10.1016/S0169-5347(03)00069-seven.

Further reading [edit]

  • Rhoades R, Pflanzer RG (1996). "Chapter 27: Regulation of Body Temperature (Clinical Focus: Pathogenesis of Fever)". Homo Physiology (3rd ed.). Philadelphia, PA: Saunders Higher. ISBN9780030051593 . Retrieved 2 April 2020. {{cite book}}: CS1 maint: uses authors parameter (link)

External links [edit]

  • Fever and Taking Your Child's Temperature
  • US National Institute of Health factsheet
  • Drugs most unremarkably associated with the adverse event Pyrexia (Fever) as reported the FDA
  • Fever at MedlinePlus
  • Why are Nosotros Then Afraid of Fevers? at The New York Times

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Source: https://en.wikipedia.org/wiki/Fever

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